[1] Lv S, Rong J, Ren S, et al. Epidemiology and diagnosis of viral myocarditis[J]. Hellenic J Cardiol, 2013, 54(5):382-391. [2] Wei J, Gao DF, Wang H, et al. Impairment of myocardial mitochondria in viral myocardial disease and its reflective win-dow in peripheral cells[J]. PLoS One, 2014, 9(12):e116239. [3] 谭振宇, 杨大浩, 廖春燕. 主动脉内球囊反搏对急性重症心肌炎心力衰竭患者室性心律失常的影响[J]. 岭南心血管病杂志, 2013, 19(5):565-567. [4] 李歆跃, 杨巍. 心脏重构中微RNA的作用[J]. 医学综述, 2015, 21(2):582-584. [5] Lok SI, de Jonge N, van Kuik J, et al. MicroRNA Expres-sion in Myocardial Tissue and Plasma of Patients with End-Stage Heart Failure during LVAD Support:Comparison of Continuous and Pulsatile Devices[J]. PloS One, 2015, 10(10):e0136-404. [6] 赵明, 单晓彤, 赵亚红, 等. 阿霉素损伤心肌细胞miRNA-378和miRNA-378* 与calumenin, GRP78, bax及bal-2相关性研究[J]. 临床心血管病志, 2016, 32(6):603-606 [7] Zhou X, Xin Q, Wang Y, et al. Total Flavonoids of Astra-galus Plays a Cardioprotective Role in Viral Myocarditis[J]. Acta Cardiol Sin, 2016, 32(1):81-88. [8] 巴金华, 窦忠霞, 柳文清, 等. 黄芪总黄酮对柯萨奇B-3病毒所致病毒性心肌炎小鼠心肌病理改变和超微结构的影响[J]. 中国心血管病究, 2013, 11(11):910-912. [9] Nagalingam RS, Sundaresan NR, Noor M, et al. Deficiency of cardiomyocyte-specific microRNA-378 contributes to the de-velopment of cardiac fibrosis involving a transforming growth fa ctorβ(TGFβ1)-dependent paracrine mechanism[J]. J Bi-ol Chem, 2014, 289(39):27199-27214. [10] Nagalingam RS, Sundaresan NR, Gupta MP, et al. A car-diac-enriched microRNA, miR-378, blocks cardiac hypertro-phy by targeting Ras signaling[J]. J Biol Chem, 2013, 288(16):11216-11232. [11] Ooi JY, Bernardo BC, McMullen JR. The therapeutic poten-tial of miRNAs regulated in settings of physiological cardiac hypertrophy[J]. Future Med Chem, 2014, 6(2):205-222. |