[1] |
丁 真, 李劲涛, 吴水才, 等. 电磁辐射的细胞生物学效应研究进展 [J]. 北京生物医学工程, 2015, 34(5): 533-537.
|
[2] |
Bellés MG, Onzalo S, Serra N, et al. Environmental exposure to low-doses of ionizing radiation. Effects on early nephr otoxicity in mice [J]. Environ Res, 2017, 156(7): 291-296.
|
[3] |
吕瑞涛. 二氧化铈纳米颗粒对大鼠心肌缺血再灌注损伤的影响 [D]. 郑州大学, 2014.
|
[4] |
莫与琳, 杨亚军, 崔 燎. TXNIP介导的氧化应激在疾病中的作用机制 [J]. 中国药理学通报, 2018, 34(1): 16-19.
|
[5] |
Feng LF, Zhang LL. Resveratrol suppresses Aβ-induced microglial activation through the TXNIP/TRX/NLRP3 signaling pathway [J]. DNA Cell Biol, 2019, 38(8): 874-879.
|
[6] |
Qu YZ, Li M, Zhao YL, et al. Astragaloside IV scerebralis-chemia-repefusion induced increase in permeability of the blood—brain hairier in rats [J]. Eur J Pharmacol, 2009, 606(1-3): 137-141.
|
[7] |
Zhang WD, Chen H, Zhang C, et al. Astragaloside IV from Astragalus membranaceus shows cardioproteetion during myocardial ischemia in vivo and in vitro [J].Planta Med, 2006, 72(1): 4-8.
|
[8] |
王 东. 松多酚微球的制备及其对60 Co-γ射线辐射防护作用的研究 [D]. 哈尔滨工业大学, 2015.
|
[9] |
唐 博. 放射性脑损伤模型的建立及损伤对神经组织的影响 [D]. 北京理工大学, 2015.
|
[10] |
朱晓婷, 陈志强. 益气化湿通络方对实验性肾功能衰竭大鼠肾脏氧化应激损伤及纤维化的改善作用 [J]. 中国应用生理学杂志, 2020, 36(1): 67-72.
|
[11] |
胡 戈, 曹 卉, 周海涛, 等. 姜黄素对过度训练大鼠肾脏细胞凋亡的调控作用及其机制 [J]. 中国应用生理学杂志, 2018, 34(6): 513-518.
|
[12] |
Liu X, Zhang J, Wang SB, et al. Astragaloside IV attenuates the H2O2-induced apoptosis of neuronal cells by inhibiting α-synuclein expression via the p38 MAPK pathway [J]. Int J Mol Med, 2017, 40(6): 1772-1780.
|
[13] |
Gui DK, Guo YP, Wang F, et al. Astragaloside IV, a novel antioxidant, prevents glucose-induced podocyte apoptosis in vitro and in vivo [J]. PLoS One, 2012, 7(6): e39824.
|
[14] |
Liu X, Shang SY, Chu WW, et al. Astragaloside IV ameliorates radiation-induced senescence via antioxidative mechanism [J]. J Pharm Pharmacol, 2020, 72: 1110-1118.
|
[15] |
Singh LP. Thioredoxin interacting protein (TXNIP) and pathogenesis of diabetic retinopath [J]. J Clin Exp Ophthalmol, 2013, 4(8): 1-11.
|
[16] |
Spindel ON, World C, Berk BC. Thioredoxin interacting protein: redox dependent and independent regulatory mechanisms [J]. Antioxid Redox Signal, 2012, 16(6): 587-96.
|
[17] |
Wei JL, Wang HR, Wang HH, et al. The role of NLRP3 inflammasome activation in radiation damage [J]. Biomed Pharmacother, 2019, 118(10): 109217.
|
[18] |
Wen Y, Liu YR, Tang TT, et al. mROS-TXNIP axis activates NLRP3 inflammasome to mediate renal injury during ischemic AKI [J]. Int J Biochem Cell Biol, 2018, 98(5): 43-53.
|
[19] |
韩晓鹏. 大鼠肾脏机械损伤早期TNF及其受体的表达 [D]. 兰州大学, 2008.
|
[20] |
Gu CM, Liu SM, Wang HY, et al. Role of the thioredoxin interacting protein in diabetic nephropathy and the mechanism of regulating NOD like receptor protein 3 inflammatory corpuscle [J]. Int J Mol Med, 2019, 43: 2440-2450.
|
[21] |
Deng HH, Chen FZ, Wang YH, et al. The role of activated NLRP3 inflammatory body in acute kidney injury in rats caused by sepsis and NLRP3-TXNIP signaling pathway [J]. Saudi J Biol Sci, 2020, 27: 1251-1259.
|
[22] |
Shan Q, Zheng GH, Han XR, et al. Troxerutin protects kidney tissue against BDE-47-induced inflammatory damage through CXCR4-TXNIP/NLRP3 signaling [J]. Oxid Med Cell Longev, 2018, 2018: 9865495.
|