[1] Knudsen JR, Steenberg DE, Hingst JR, et al. Prior exercise in humans redistributes intramuscular GLUT4 and enhances insulin-stimulated sarcolemmal and endosomal GLUT4 translocation[J]. Mol Metab, 2020, 39(39): 100998-101007. [2] Kohn AD, Summers SA, Birnbaum MJ, et al. Expression of a constitutively active Akt Ser/Thr kinase in 3T3-L1 adipocytes stimulates glucose uptake and glucose transporter 4 translocation[J]. J Biol Chem, 1996, 271(49): 31372-31378. [3] Cartee GD. Roles of TBC1D1 and TBC1D4 in insulin-and exercise-stimulated glucose transport of skeletal muscle[J]. Diabetologia, 2015, 58(1): 19-30. [4] Zhang C, Jiang Y, Liu J, et al. AMPK/AS160 mediates tiliroside derivatives-stimulated GLUT4 translocation in muscle cells[J]. Drug Des Dev Ther, 2018, 12(12): 1581-1587. [5] Koh JH, Hancock CR, Han DH, et al. AMPK and PPARβ positive feedback loop regulates endurance exercise training-mediated GLUT4 expression in skeletal muscle[J]. Am J Physiol Endoc M, 2019, 316(5): 931-939. [6] Rubinstein MP, Kovar M, Purton JF, et al. Converting IL-15 to a superagonist by binding to soluble IL-15Rα[J]. Proc Natl Acad Sci USA, 2006, 103(24): 9166-9171. [7] Zhang T, Qi Z, Wang H, et al. Adeno-associated virus-mediated knockdown of SLC16A11 improves glucose tolerance and hepatic insulin signaling in high fat diet-fed mice[J]. Exp Clin Endocr Diab, 2021, 129(2): 104-111. [8] Han R, Qiu H, Zhong J, et al. Si Miao Formula attenuates non-alcoholic fatty liver disease by modulating hepatic lipid metabolism and gut microbiota[J]. Phytomedicine, 2021, 85(85): 153544-153556. [9] Song H, Guan Y, Zhang L, et al. SPARC interacts with AMPK and regulates GLUT4 expression[J]. Biochem Bioph Res Co, 2010, 396(4): 961-966. [10] Nadeau L, Aguer C. Interleukin-15 as a myokine: mechanistic insight into its effect on skeletal muscle metabolism[J]. Appl Physiol Nutr Me, 2019, 44(3): 229-238. [11] Busquets S, Figueras M, Almendro V, et al. Interleukin-15 increases glucose uptake in skeletal muscle An antidiabetogenic effect of the cytokine[J]. Biochim Biophys Acta, 2006, 1760(11): 1613-1617. [12] Sun H, Liu D. Hydrodynamic delivery of interleukin 15 gene promotes resistance to high fat diet-induced obesity, fatty liver and improves glucose homeostasis[J]. Gene Ther, 2015, 22(4): 341-347. [13] Krolopp JE, Thornton SM, Abbott MJ. IL-15 activates the Jak3/STAT3 signaling pathway to mediate glucose uptake in skeletal muscle cells[J]. Front Physiol, 2016, 7(7): 626-636. [14] Larance M, Ramm G, Stöckli J, et al. Characterization of the role of the Rab GTPase-activating protein AS160 in insulin-regulated GLUT4 trafficking[J]. J Biol Chem, 2005, 280(45): 37803-37813. [15] Mendes AI, Matos P, Moniz S, et al. Protein kinase WNK1 promotes cell surface expression of glucose transporter GLUT1 by regulating a Tre-2/USP6-BUB2-Cdc16 domain family member 4 (TBC1D4)-Rab8A complex[J]. J Biol Chem, 2010, 285(50): 39117-39126. [16] Sriwijitkamol A, Coletta DK, Wajcberg E, et al. Effect of acute exercise on AMPK signaling in skeletal muscle of subjects with type 2 diabetes: a time-course and dose-response study[J]. Diabetes, 2007, 56(3): 836-848. [17] Xie B, Chen Q, Chen L, et al. The inactivation of RabGAP function of AS160 promotes lysosomal degradation of GLUT4 and causes postprandial hyperglycemia and hyperinsulinemia[J]. Diabetes, 2016, 65(11): 3327-3340. [18] Stenbit AE, Tsao TS, Li J, et al. GLUT4 heterozygous knockout mice develop muscle insulin resistance and diabetes[J]. Nat Med, 1997, 3(10): 1096-1101. [19] Zheng A, Arias EB, Wang H, et al. Exercise-induced improvement in insulin-stimulated glucose uptake by rat skeletal muscle is absent in male AS160-knockout rats, partially restored by muscle expression of phosphomutated AS160, and fully restored by muscle expression of wild-type AS160[J]. Diabetes, 2022, 71(2): 219-232. [20] Brenner AK, Andersson Tvedt TH, Bruserud O. The complexity of targeting PI3K-Akt-mTOR signalling in human acute myeloid leukaemia: The importance of leukemic cell heterogeneity, neighbouring mesenchymal stem cells and immunocompetent cells[J]. Molecules, 2016, 21(11): 1512-1544. |